High dietary advanced glycation end products are associated with poorer spatial learning and accelerated Aβ deposition in an Alzheimer mouse model.

Chronic intake of AGEs is associated with higher levels of peripheral AGEs and insoluble Aβ 42 In the Tg2576 H‐AGE diet group (but not in the R‐AGE diet group), the correlation of Aβ42 in the hippocampus with urine CML approached significance ( r = 0.619; P = 0.057) and was significant with MG ( r = 0.632; P = 0.050; Table 2 ).

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