Overexpression of MARCKS increased Rac1 activity; however, this increase did not reach statistical significance (Figure 6 G).
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Myristoylated Alanine-Rich Protein Kinase Substrate (MARCKS) Regulates Small GTPase Rac1 and Cdc42 Activity and Is a Critical Mediator of Vascular Smooth Muscle Cell Migration in Intimal Hyperplasia Formation.
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