Mitochondrial Reactive Oxygen Species Mediate Cardiac Structural, Functional, and Mitochondrial Consequences of Diet-Induced Metabolic Heart Disease.

In WT mice, complex I–mediated respiration linked to maximal ATP synthesis rate (state III) and proton leak (state IV) both tended to be lower in HFHS‐fed mice but did not reach statistical significance (Figure 3 A and 3 B).

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