Glycogen Synthase Kinase 3β Inhibition Improves Myocardial Angiogenesis and Perfusion in a Swine Model of Metabolic Syndrome.

Pro‐Angiogenic Signaling Pathway There were greater expression levels of the following proangiogenic protein markers in the GSk‐3β inhibited group compared to the control group including: VEGFR1 ( P =0.046), γ‐catenin ( P =0.02), and VE‐cadherin ( P =0.01) and β‐catenin that failed to reach significance ( P =0.07).

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There were also increases in expression in ERK1/2 ( P =0.17) and eNOS ( P =0.12) in the GSK‐3β inhibited group compared to the HCC control; however, these did not reach statistical significance (Table 2 ).

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