Intensified vmPFC surveillance over PTSS under perturbed microRNA-608/AChE interaction.

Although this finding is only marginally significant, we speculated that it might indicate that greater vmPFC functioning may operate as a clinically helpful compensatory mechanism for A-allele individuals to neutralize their sensitivity to stress, and that they present no increase in PTSS owing to the recruitment of the vmPFC to overcome their stress vulnerability.

also in 5,076 other papers