Human class I major histocompatibility complex alleles determine central nervous system injury versus repair.

We found a borderline significance ( P = 0.05, rank sum test) for the increased extent of pathology in the hippocampus and striatum of Aβ 0 .β2m 0 .Hβ2m + .A11 + mice (1.656 ± 0.256, N = 32) as compared to Aβ 0 .β2m 0 .Hβ2m + .B27 + mice (0.917 ± 0.255, N = 24).

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There was a statistical trend ( P = 0.070, rank sum test) for less brain pathology in Aβ 0 .β2m 0 .Hβ2m + mice as compared to Aβ 0 .β2m 0 .Hβ2m + .A11 + mice (0.964 ± 0.137, N = 112).

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However, since the differences for brain pathology comparing Aβ 0 .β2m 0 .Hβ2m + mice did not reach statistical significance, we consider this less likely.

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