Aspergillus antigen induces robust Th2 cytokine production, inflammation, airway hyperreactivity and fibrosis in the absence of MCP-1 or CCR2.

The BAL fluid eosinophil response in MCP-1- and CCR2-deficient mice was slightly (~30–40%) smaller than in wild-type mice, but this difference did not reach statistical significance (Fig. 1 ).

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