Furthermore, the observation that the pTEPPerC1 plasmid conferred growth advantage to both pathogen and commensal strains (Figure S3 ), and that, approaching significance, the wt EPEC outcompeted the Δ perC strain when grown in conditions mimicking the human gut increased confidence that expression of PerC confers a measurable growth advantage to EPEC.
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PerC Manipulates Metabolism and Surface Antigens in Enteropathogenic <i>Escherichia coli</i>.
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