Integrating personalized gene expression profiles into predictive disease-associated gene pools.

While this difference is highly significant ( p- value <10 −77 , Mann–Whitney U test), in absolute numbers the overlap is small: while a typical asthmatic subject has on average 379 perturbed genes, the average number of shared perturbed genes between two asthmatic subjects is only 24 (Fig. 2d ).

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As shown in Supplementary Table 1 , group-wise DE genes cover only a small fraction of the asthma-related pathways: only 7 out of 35 pathways show nominally significant enrichment (uncorrected p -value < 0.05, Fisher’s exact test); after Bonferroni correction, only two pathways remain.

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