Inhibition of neuronal cell death and retinal damage dependent MG proliferation was efficient with either ZVDL or Nec1+ ZVAD, which both reduced CASP8 expression and showed a trend increase in RIPK3, a regulator of RIPK1 activation and inhibitor of CASP8-mediated compensatory proliferation in other systems 73 .
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Retinal cell death dependent reactive proliferative gliosis in the mouse retina.
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