BRD2/3/4 mRNA expression was relative lower in SPOP-mutated tumors than that in SPOP-WT specimens in our cohorts, although the difference did not reach statistical significance (except BRD2 ) ( Supplementary Fig. 2c ).
← all excerpts
Intrinsic BET inhibitor resistance in SPOP-mutated prostate cancer is mediated by BET protein stabilization and AKT-mTORC1 activation.
1
—
—