TLR2 stimulation impairs anti-inflammatory activity of M2-like macrophages, generating a chimeric M1/M2 phenotype.

Arthritis Res Ther · 2017 · PMC5667453 · PMID 29096690

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In contrast, TLR4 ligand LPS or activation by IFN-γ/LPS showed a trend toward lower MFI of M2-polarized markers CD14, CD163, and CD206 while increasing the signal for M1-specific marker CD86, thus underlining the role of CD86 as a marker of “activated” M1 macrophages (Additional file 2 ).

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