Hypothermic machine perfusion ameliorates inflammation during ischemia‑reperfusion injury via sirtuin‑1‑mediated deacetylation of nuclear factor‑κB p65 in rat livers donated after circulatory death.

Notably, the bile flow production was marginally higher in the HMP group compared with the CS group, however, this did not reach statistical significance after 120 min reperfusion (9.29±2.48 vs. 7.90±2.36 μl/h/g liver; P=0.704).

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