Doxycycline inhibits experimental cerebral malaria by reducing inflammatory immune reactions and tissue-degrading mediators.

After DOX treatment, the capacity of brain microvessels to cross-present PbA-derived antigen to TCR-transduced NFAT-lacZ reporter cells was slightly decreased compared to that from untreated PbA infected animals, but did not reach statistical significance (Figure G in S2 Fig ).

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