ES2-N cells showed a trend similar to ES2-A, in all conditions, with the exception of ES2-NH, in which there was a tendency for higher cell death levels upon carboplatin exposure, though not statistically significant (Fig. 4a and Table 4 A).
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Cysteine boosters the evolutionary adaptation to CoCl<sub>2</sub> mimicked hypoxia conditions, favouring carboplatin resistance in ovarian cancer.
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