Bifidobacterium breve MRx0004 protects against airway inflammation in a severe asthma model by suppressing both neutrophil and eosinophil lung infiltration.

These pro-inflammatory mediators are known to be elevated in T H 2-low asthmatic patients and although readouts failed to reach statistical significance in the current model, the suppression of these cytokines may provide insight into the mechanism by which MRx0004 elicits its effect.

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