A Single Primary Blast-Induced Traumatic Brain Injury in a Rodent Model Causes Cell-Type Dependent Increase in Nicotinamide Adenine Dinucleotide Phosphate Oxidase Isoforms in Vulnerable Brain Regions.

Immunoblot analysis of 4HNE products identified two major bands corresponding to molecular weights of 70 and 100 kDa, which showed a trend toward an increase in cerebral hemispheres, hippocampus, thalamus, and cerebellum; however, such changes were not significant ( Fig. 9 ).

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