Loss of <i>Mecp2</i> Causes Atypical Synaptic and Molecular Plasticity of Parvalbumin-Expressing Interneurons Reflecting Rett Syndrome-Like Sensorimotor Defects.

When we analyzed presymptomatic Mecp2 KO mice, we found a trend toward decreased maximum amplitudes of VSD signals in S1 slices from mutant mice compared with WT slices which did not reach statistical significance ( Fig. 4 G ).

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