NFκB Inhibition Mitigates Serum Amyloid A-Induced Pro-Atherogenic Responses in Endothelial Cells and Leukocyte Adhesion and Adverse Changes to Endothelium Function in Isolated Aorta.

Results Consistent with previous data indicating that SAA increased VEGF expression in primary endothelial cells [ 22 ], cultured HCtAEC supplemented with 10 μg/mL SAA showed ~2-fold increase in secretory levels of VEGF relative to control cells ( Figure 1 ), albeit this did not reach statistical significance.

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