On the other hand, at the postsynaptic level, EtOH withdrawal induced an increased expression of the AMPA subunit GluA1 and of the metabotropic Glu5 receptors, while chronic EtOH exposure showed a trend toward an increased expression without reaching a statistical significance (Figures 5B,F ) of the same proteins.
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Glutamate Receptor-Mediated Neurotoxicity in a Model of Ethanol Dependence and Withdrawal in Rat Organotypic Hippocampal Slice Cultures.
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