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Spatiotemporal expression and inhibition of prolyl oligopeptidase contradict its involvement in key pathologic mechanisms of kainic acid-induced temporal lobe epilepsy in rats.

Epilepsia Open · 2019 · PMC6398098 · PMID 30868119

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Accordingly, the OX‐42% area and CD‐68 scores remained significantly higher in both SE groups compared to the control animals, except in the dentate hilus (DH) region where it did not reach statistical significance.

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