albicans strain SC5314 through the murine kidney, showed that despite leading to increased phenotypic variability within the population possibly by microevolution, the overall virulence, fungal fitness and the host response did not follow a clear trend between infected animals, revealing that the C. albicans strain used was already well adapted to the murine kidney [ 124 ].
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Microevolution of the pathogenic yeasts <i>Candida albicans</i> and <i>Candida glabrata</i> during antifungal therapy and host infection.
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