Endothelial Cells Regulate Physiological Cardiomyocyte Growth via VEGFR2-Mediated Paracrine Signaling.

34 Although both AAV-ErbB4ECD and afatinib inhibited the hypertrophy-associated biochemical signals induced by VEGF-B in the heart, their effects on the VEGF-B–induced increase in cardiac mass did not reach statistical significance ( Figure XC through XH in the online-only Data Supplement ).

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