Distinct Phenotypic and Genomic Signatures Underlie Contrasting Pathogenic Potential of <i>Staphylococcus epidermidis</i> Clonal Lineages.

A/C cluster isolates almost equally originated from infection ( n = 28/59, 47%) and colonization ( n = 31/59, 53%) sources, while B cluster isolates were mostly from colonization (17/23, 70%) ( p = 0.127) even though this different distribution did not reach statistical significance.

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