Our current data also shows that unlike p-Src and p-ERK1/2, there is a clear trend for an increase in p-EGFR in the mucosal side of the airways, which is in-line with findings demonstrating increased EGFR immunoreactivity in the bronchial epithelium [ 25 ].
← all excerpts
Ang-(1-7)/ MAS1 receptor axis inhibits allergic airway inflammation via blockade of Src-mediated EGFR transactivation in a murine model of asthma.
1
—
—