Tobacco-Derived Lipopolysaccharide, Not Microbial Translocation, as a Potential Contributor to the Pathogenesis of Rheumatoid Arthritis.

LBP also showed a trend towards correlating with DAS 28-3 in the subgroup of RA patients that used tobacco products ( r = 0.4599; p = 0.0731), while a moderate correlation between serum SP-D and disease duration ( r = 0.4181; p = 0.0073), as well as weak negative correlations of this biomarker with I-FABP ( r = −0.2869; p = 0.0123) and LPS ( r = −0.3287; p = 0.0411) were also noted. 3.2.

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