Subsequently, a clear trend showing suppressed mitochondrial respiration could be identified by measuring these mitochondrial energetic factors, further suggesting that compromised integrity of the electron transport chain plays a significant role in the production of ROS and subsequent cellular injury.
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<i>N</i>-Acetyl cysteine ameliorates hyperglycemia-induced cardiomyocyte toxicity by improving mitochondrial energetics and enhancing endogenous Coenzyme Q<sub>9/10</sub> levels.
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