Selenomethionine supplementation reduces lesion burden, improves vessel function and modulates the inflammatory response within the setting of atherosclerosis.

On a normal chow diet ApoE −/- mice develop plaques both within the aortic root and along the length of the aorta, which are reduced by ~50% in mice cohorts fed 2 mg/kg SeMet over a period of 12 weeks, although this did not reach statistical significance.

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