Inhibition of histone deacetylation rescues phenotype in a mouse model of Birk-Barel intellectual disability syndrome.

Interestingly, a clear trend toward impaired working memory was also observed in shRNAmir- Kcnk9 injected animals (Fig. 2e ), suggesting that Kcnk9 expression also controls working memory-related activity of LC neurons.

also in 19,460 other papers