Phosphorylated tau at residues Ser202/Thr205 was unchanged in the soluble fraction but showed a trend to significance in crude synaptosomes of the miR‐181a inhibitor group (Figure 4 k‐L).
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miR-181a negatively modulates synaptic plasticity in hippocampal cultures and its inhibition rescues memory deficits in a mouse model of Alzheimer's disease.
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