It has been shown to be associated with increased levels of AEA, an endocannabinoid which also binds to PPARs, in the basolateral amygdala (BLA) [ 32 ] and in the dorsolateral periaqueductal grey (dlPAG) [ 31 ] and a strong trend for increased tissue levels of PEA and OEA, endogenous ligands of PPARs, in the BLA [ 7 , 8 ].
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Pharmacological Blockade of PPAR Isoforms Increases Conditioned Fear Responding in the Presence of Nociceptive Tone.
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