High-Density Lipoprotein (HDL) Inhibits Serum Amyloid A (SAA)-Induced Vascular and Renal Dysfunctions in Apolipoprotein E-Deficient Mice.

Consistent with increased immune reactivity for VCAM-1, the gene expression of VCAM-1 in the SAA-treated mice increased 50% (reaching ~2.1-fold higher than in the corresponding vehicle control), although this did not reach statistical significance.

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