On the other hand, though in vivo treatment of AGI decreased levels of ex vivo insulin secretion under high glucose conditions in primary islets (basal insulin secretion showed a trend of decrement with no significance) (Fig. 5f ), it did not affect ex vivo GIIS responses (Fig. 5g ) as compared with islets from untreated wild-type mice.
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The glucose-lowering effects of α-glucosidase inhibitor require a bile acid signal in mice.
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