Conditioned media from FM-MA or PR8 infections caused moderate increases in HLA-A/B/C levels, whereas media from NS1 mutant virus infections elicited striking, statistically significant increases in HLA-A/B/C and showed a trend toward increased HLA-B and HLA-E levels when measured independently ( Fig. 8B , bottom).
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Defective Influenza A Virus RNA Products Mediate MAVS-Dependent Upregulation of Human Leukocyte Antigen Class I Proteins.
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