Predators can influence the host-parasite dynamics of their prey via nonconsumptive effects.

Our linear model that included an interaction between predator cue treatment and infection status did not detect any significant predictors of offspring number, though microsporidian infection had a nearly significant and negative effect (−6.368 ± 3.600 SE ; p = .079), while infection in the sustained predator cue treatment had a nearly significant and positive effect (8.364 ± 5.008; p = .097), suggesting that microsporidian infection in the absence of predator cues resulted in low offspring production (Figure 3 ).

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