Homeostatic plasticity fails at the intersection of autism-gene mutations and a novel class of common genetic modifiers.

The percent PHP expression is decreased from 152% in the ASH1L /+ mutant, to 114% in the double heterozygote, which is not different from baseline (p=0.2) and represents a highly significant suppression (p<0.01) compared to the ASH1L /+ mutant alone (p<0.01) ( Figure 1—figure supplement 2B ).

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The data actual shows an increase in bouton number for the CHD8/+;PPP2R5D/+ genotype compared to wildtype and PPP2R5D/+ alone, it did not reach statistical significance compared to CHD8/+ alone but perhaps this is due to the sample size?

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