MiR-181c-5p Promotes Inflammatory Response during Hypoxia/Reoxygenation Injury by Downregulating Protein Tyrosine Phosphatase Nonreceptor Type 4 in H9C2 Cardiomyocytes.

In normoxia group, there was a trend towards an increased expression of phosphorylation of I κ B α (Ser 32/36 ) in miR-181c-5p antagomir-transfected H9C2 cells, which however did not reach statistical significance ( Figure 3(b) ).

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