Mitochondrial damage and senescence phenotype of cells derived from a novel frataxin G127V point mutation mouse model of Friedreich's ataxia.

Finally, although the proportion of apoptotic cells was higher in MUT than in WT and HET MEF populations, the difference did not reach statistical significance (MUT, 7.7±1.1%; WT, 5.5±2.7%; and HET, 3.5±0.4%).

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