The mig1 mig2 double mutations upregulated ATG39 induction upon exposure to tunicamycin ( S7A Fig ), although the difference in ATG39 expression levels between wild-type and mig1 mig2 double mutant cells did not reach statistical significance when DTT was used as an ER stressor ( Fig 4C ).
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Snf1 AMPK positively regulates ER-phagy via expression control of Atg39 autophagy receptor in yeast ER stress response.
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