Consequently, NAS was significantly elevated in the HFD groups as compared with the control groups in both genotypes and showed a trend to be higher in the Cx32ΔTg-HFD than in the Wt-HFD group (Fig.
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A novel model of non-alcoholic steatohepatitis with fibrosis and carcinogenesis in connexin 32 dominant-negative transgenic rats.
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