p47^phox deficiency improves cognitive impairment and attenuates tau hyperphosphorylation in mouse models of AD.

Although soluble Aβ 1–42 showed a trend of decrease in APP/PS1 - Ncf1 −/− mice compared with APP/PS1 mice, the difference was not statistically significant (Fig. 2 h).

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APP/PS1 mice aged 9 months and 12 months displayed a decreasing trend in fall latency compared with WT mice (Additional file 1 : Fig.

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