Endoplasmic Reticulum (ER) Stress-Generated Extracellular Vesicles (Microparticles) Self-Perpetuate ER Stress and Mediate Endothelial Cell Dysfunction Independently of Cell Survival.

Front Cardiovasc Med · 2020 · PMC7758248 · PMID 33363219

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On the other hand, while TG caused a trend to increase in the phosphorylation of p38 MAPK ( Figure 10C ), TG-generated MPs showed a trend for decrease ( Figure 10D ).

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