Additionally, knockdown of DNM2 also significantly increased the proportion of late apoptotic cells in the presence of bortezomib and sEVs, and CLTC knockdown induced the similar effect with a trend close to significance (Figure 6 J-K).
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Endocytic pathway inhibition attenuates extracellular vesicle-induced reduction of chemosensitivity to bortezomib in multiple myeloma cells.
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