CD73 contributes to anti-inflammatory properties of afferent lymphatic endothelial cells in humans and mice.

Furthermore, we found highly significant changes in MHC class II (upregulation), CD40 (upregulation), CD83 (upregulation), CD86 (upregulation), CD80 (downregulation), as well as in the adenosine receptor A2b (downregulation) on DCs after they had been co‐cultured with siCD73‐silenced or CRISPR/Cas9 KO LECs (Fig. 3E, F, and H ).

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However, immature moDCs bound significantly more to TNF‐α‐treated LECs and there was a strong trend for increased binding to silenced LECs under these conditions (Fig. 3D ).

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