All markers showed a trend towards reduced expression in early disease (fatty streak lesions) and calcified arteries compared with healthy, with the lowest expression in calcified arteries (Table I in the Data Supplement ; Figure 1 A).
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Endoplasmic Reticulum Stress Mediates Vascular Smooth Muscle Cell Calcification via Increased Release of Grp78 (Glucose-Regulated Protein, 78 kDa)-Loaded Extracellular Vesicles.
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