Consistent with previous reports, the growth of Wa and Gottfried strains was sialidase-insensitive or even showed a trend for a slight increase of infectivity following sialidase treatment (Figs 7A and S1 , respectively), while replication of OSU was inhibited by sialidase treatment ( Fig 7B ).
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Infection of porcine small intestinal enteroids with human and pig rotavirus A strains reveals contrasting roles for histo-blood group antigens and terminal sialic acids.
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