Targeting natural splicing plasticity of APOBEC3B restricts its expression and mutagenic activity.

In LUAD, the correlations were comparable for both A3A1 ( P = 3.12E−12; rho = 0.31) and A3B1 ( P = 7.73E−12; rho = 0.30) and in LUSC—the correlation was moderately significant only for A3A1 ( P = 1.86E−02; rho = 0.17) (Fig. 3a, b ).

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