Improvement of ECM-based bioroot regeneration via N-acetylcysteine-induced antioxidative effects.

1 b, the survival of the hDFCs showed a trend of first increasing and then decreasing, and the survival significantly decreased following treatment with concentrations of H 2 O 2 above 100 μM; this result was consistent with previous studies showing that high ROS levels cause cellular damage and dysfunction, but low basal ROS levels are necessary and advantageous for maintaining cellular proliferation, differentiation, and survival [ 27 ].

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