Isoliquiritigenin Reverses Epithelial-Mesenchymal Transition Through Modulation of the TGF-β/Smad Signaling Pathway in Endometrial Cancer.

Similarly, TGF-β1 significantly reduced the expression of E-cadherin in Ishikawa cells ( p < 0.05, Figure 4 a–c), but the increase in N-cadherin expression failed to reach statistical significance.

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The difference between two means was considered statistically significant when p < 0.05 and highly significant when p < 0.01. 5.

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The results of western blotting show that 10 μM ISL significantly reversed the increased protein expression of N-cadherin in HEC-1A cells ( p < 0.05, Figure 3 c) and upregulated the protein expression of E-cadherin in Ishikawa cells ( p < 0.05, Figure 4 b), while the protein expression of E-cadherin after ISL treatment showed an increasing trend in HEC-1A cells ( Figure 3 b), and the protein expression of N-cadherin after ISL treatment showed a downward trend in Ishikawa cells ( Figure 4 c) without statistical significance.

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