Demonstrations of strong associations between eosinophil numbers and mycobacterial and hookworm infections suggest that the eosinophil contribution to bystander immunity is likely to be significant ( O'Shea et al., 2018 ).
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Il4ra-independent vaginal eosinophil accumulation following helminth infection exacerbates epithelial ulcerative pathology of HSV-2 infection.
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The sentences
Flow cytometry analysis at this time point showed a trend for reduced numbers of ILC2s and significantly less IL-5-producing ILC2s in the FGT of HpARI-treated Nb + HSV-2 mice compared with untreated counterparts ( Figures S7 B and S7C).